| 000 | 06144naaaa2201477uu 4500 | ||
|---|---|---|---|
| 001 | https://directory.doabooks.org/handle/20.500.12854/76898 | ||
| 005 | 20220714164334.0 | ||
| 020 | _abooks978-3-0365-1995-1 | ||
| 020 | _a9783036519944 | ||
| 020 | _a9783036519951 | ||
| 024 | 7 |
_a10.3390/books978-3-0365-1995-1 _cdoi |
|
| 041 | 0 | _aEnglish | |
| 042 | _adc | ||
| 072 | 7 |
_aGP _2bicssc |
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| 100 | 1 |
_aLee, Bae Hwan _4edt _91579728 |
|
| 700 | 1 |
_aLee, Bae Hwan _4oth _91579728 |
|
| 245 | 1 | 0 | _aNeuroprotection: Rescue from Neuronal Death in the Brain |
| 260 |
_aBasel, Switzerland _bMDPI - Multidisciplinary Digital Publishing Institute _c2021 |
||
| 300 | _a1 electronic resource (408 p.) | ||
| 506 | 0 |
_aOpen Access _2star _fUnrestricted online access |
|
| 520 | _aDear Colleagues, The brain is vulnerable to injury. Following injury in the brain, apoptosis or necrosis may occur easily, leading to various functional disabilities. Neuronal death is associated with a number of neurological disorders including hypoxic ischemia, epileptic seizures, and neurodegenerative diseases. The brain subjected to injury is regarded to be responsible for the alterations in neurotransmission processes, resulting in functional changes. Oxidative stress produced by reactive oxygen species has been shown to be related to the death of neurons in traumatic injury, stroke, and neurodegenerative diseases. Therefore, scavenging or decreasing free radicals may be crucial for preventing neural tissues from harmful adversities in the brain. Neurotrophic factors, bioactive compounds, dietary nutrients, or cell engineering may ameliorate the pathological processes related to neuronal death or neurodegeneration and appear beneficial for improving neuroprotection. As a result of neuronal death or neuroprotection, the brain undergoes activity-dependent long-lasting changes in synaptic transmission, which is also known as functional plasticity. Neuroprotection implying the rescue from neuronal death is now becoming one of global health concerns. This Special Issue attempts to explore the recent advances in neuroprotection related to the brain. This Special Issue welcomes original research or review papers demonstrating the mechanisms of neuroprotection against brain injury using in vivo or in vitro models of animals as well as in clinical settings. The issues in a paper should be supported by sufficient data or evidence. Prof. Bae Hwan Lee Guest Editor | ||
| 540 |
_aCreative Commons _fhttps://creativecommons.org/licenses/by/4.0/ _2cc _4https://creativecommons.org/licenses/by/4.0/ |
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| 546 | _aEnglish | ||
| 650 | 7 |
_aResearch & information: general _2bicssc _9928234 |
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| 653 | _aglobal cerebral ischemia | ||
| 653 | _aamiloride | ||
| 653 | _asodium-hydrogen exchanger-1 | ||
| 653 | _azinc | ||
| 653 | _aneuronal death | ||
| 653 | _aneuroprotection | ||
| 653 | _aneurodegenerative disorder | ||
| 653 | _acholine acetyltransferase (ChAT) | ||
| 653 | _atrimethyltin (TMT) | ||
| 653 | _abean phosphatidylserine (Bean-PS) | ||
| 653 | _abrain-derived neurotrophic factor | ||
| 653 | _amoderate hypoxia | ||
| 653 | _aphysical exercise | ||
| 653 | _apsychomotor function | ||
| 653 | _areaction time | ||
| 653 | _acortisol | ||
| 653 | _acatecholamines | ||
| 653 | _anitrite | ||
| 653 | _aendotheline-1 | ||
| 653 | _alactate | ||
| 653 | _apyridoxine deficiency | ||
| 653 | _aischemia | ||
| 653 | _agerbil | ||
| 653 | _ahomocysteine | ||
| 653 | _acell death | ||
| 653 | _aglia | ||
| 653 | _aneurogenesis | ||
| 653 | _aN-acetyl-l-cysteine | ||
| 653 | _atransient receptor potential melastatin 2 | ||
| 653 | _aneurodegeneration | ||
| 653 | _aAlzheimer's disease | ||
| 653 | _ametabolic disease | ||
| 653 | _aadiponectin | ||
| 653 | _ainsulin | ||
| 653 | _aantioxidants | ||
| 653 | _astroke | ||
| 653 | _apreventive gene therapy | ||
| 653 | _aadenoviral vector | ||
| 653 | _aVEGF | ||
| 653 | _aGDNF | ||
| 653 | _aNCAM | ||
| 653 | _ahuman umbilical cord blood mononuclear cells | ||
| 653 | _aantioxidant | ||
| 653 | _abrain | ||
| 653 | _aneurodegenerative disease | ||
| 653 | _aoxidative stress | ||
| 653 | _aPGC-1α | ||
| 653 | _avascular endothelial growth factor | ||
| 653 | _avascular endothelial growth factor receptor 2 | ||
| 653 | _aPI3K/AKT | ||
| 653 | _aMEK/ERK | ||
| 653 | _astatus epilepticus | ||
| 653 | _ahippocampus | ||
| 653 | _amiddle cerebral artery occlusion | ||
| 653 | _areperfusion injury | ||
| 653 | _alipid emulsion | ||
| 653 | _aexcitotoxicity | ||
| 653 | _aapoptosis | ||
| 653 | _aGPR4 receptor | ||
| 653 | _aMPP+ | ||
| 653 | _aParkinson's disease | ||
| 653 | _aCRISPR/cas9 | ||
| 653 | _aischemic stroke | ||
| 653 | _ablood brain barrier | ||
| 653 | _ananoparticle-based drug delivery | ||
| 653 | _abrain targeting | ||
| 653 | _aBDNF | ||
| 653 | _amiRNAs | ||
| 653 | _asynaptic plasticity | ||
| 653 | _adepression | ||
| 653 | _aglioblastoma | ||
| 653 | _aastrocytes | ||
| 653 | _aastrocytic networks | ||
| 653 | _aconnexin 43 | ||
| 653 | _acalcium activity | ||
| 653 | _aneural injury | ||
| 653 | _animodipine | ||
| 653 | _asubarachnoid haemorrhage | ||
| 653 | _aacid-sensing ion channels | ||
| 653 | _aoxygen-glucose deprivation | ||
| 653 | _aliver growth factor | ||
| 653 | _ainflammation | ||
| 653 | _amicroglia | ||
| 653 | _aTg2576 transgenic mice | ||
| 653 | _aamyloid-beta | ||
| 653 | _aoculomotor system | ||
| 653 | _atrophic factors | ||
| 653 | _amotoneurons | ||
| 653 | _aaxotomy | ||
| 653 | _aamyotrophic lateral sclerosis | ||
| 653 | _aelectroneutral transport | ||
| 653 | _acation-chloride cotransporters | ||
| 653 | _aKCCs | ||
| 653 | _aNKCCs | ||
| 653 | _aWNK-SPAK/OSR1 | ||
| 653 | _aascorbic acid | ||
| 653 | _aaging | ||
| 653 | _aorganotypic hippocampal slice culture | ||
| 653 | _an/a | ||
| 856 | 4 | 0 |
_awww.oapen.org _uhttps://mdpi.com/books/pdfview/book/4368 _70 _zDOAB: download the publication |
| 856 | 4 | 0 |
_awww.oapen.org _uhttps://directory.doabooks.org/handle/20.500.12854/76898 _70 _zDOAB: description of the publication |
| 999 |
_c2984926 _d2984926 |
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