Host Immune Responses and Pathogenesis to Brucella spp. Infection
Material type:![Article](/opac-tmpl/lib/famfamfam/AR.png)
- books978-3-0365-2419-1
- 9783036524184
- 9783036524191
- Research & information: general
- Recombinant vaccine
- divalent vaccine
- brucellosis
- Omp25
- L7/L12
- Brucella abortus 544
- ST2 receptor
- Brucella abortus
- oral infection
- human endometrial cells
- internalization
- intracellular replication
- decidualization
- chemokines
- macrophages
- Brucella
- HSC
- MHC
- IL-10
- cell cycle
- (p)ppGpp
- rsh
- neurobrucellosis
- platelets
- brain microvascular endothelial cells
- endothelial cells
- adhesins
- Ig-like domain
- monomeric autotransporters
- trimeric autotransporters
- extracellular matrix
- polar localization
- virulence factors
- vaccine candidates
- fibronectin
- canonical inflammasome
- non-canonical inflammasome
- NLR
- pyroptosis
- ASC
- caspase-11
- caspase-1
- IL-1β
- gDNA
- replication niche
- reservoir
- persistence
- survival
- chronic infection
- n/a
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OPJGU Sonepat- Campus | E-Books Open Access | Available |
Open Access star Unrestricted online access
Brucellosis, caused by the facultative intracellular bacteria Brucella species, is one the most prevalent zoonosis worldwide. • The articles described in this book report several aspects of host-Brucella interactions. • The findings described here will help to advance in the comprehension of bacterial pathogenesis and contribute to the future development of drugs or vaccines to control brucellosis.
Creative Commons https://creativecommons.org/licenses/by/4.0/ cc https://creativecommons.org/licenses/by/4.0/
English
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