Cellular response to physical stress and therapeutic applications / Tadamichi Shimizu and Takashi Kondo, editors.

Includes bibliographical references and index.

Description based on print version record.

CELLULAR RESPONSE TO PHYSICAL STRESS AND THERAPEUTIC APPLICATIONS; CELLULAR RESPONSE TO PHYSICAL STRESS AND THERAPEUTIC APPLICATIONS; Library of Congress Cataloging-in-Publication Data; CONTENTS; PREFACE; Chapter 1: THE MECHANISMS OF RADIATION-INDUCED CELL DEATH AND ITS APPLICATION FOR CANCER THERAPY; ABSTRACT; 1. INTRODUCTION; 2. RADIATION-INDUCED CELL DEATH; 3. MECHANISMS OF RADIATION-INDUCED CELL DEATH; 4. IMPLICATIONS OF RADIATION-INDUCED CELL DEATH; 5. RADIATION-INDUCED APOPTOSIS AND THE MODIFICATIONS FOR CANCER THERAPY; CONCLUSION; ACKNOWLEDGMENTS; REFERENCES.

Chapter 2: NITRIC OXIDE-MEDIATED SIGNAL TRANSDUCTION INDUCED BY IONIZING RADIATIONABSTRACT; 1. INTRODUCTION; 2. ROLES OF NO IN RADIATION-INDUCED ADAPTIVE RESPONSE; 3. ROLES OF NO IN LOW DOSE HYPER-RADIOSENSITIVITY; 4. ROLES OF NO IN RADIATION-INDUCED BYSTANDER RESPONSE; CONCLUSION; ACKNOWLEDGMENTS; REFERENCES; Chapter 3: BIOLOGICAL EFFECTSOF ELECTROMAGNETIC FIELDS AND THEIR THERAPEUTIC APPLICATIONS; ABSTRACT; 1. INTRODUCTION; 2. RESULTS OF RESEARCH PROJECTS; 3. MECHANISMS UNDERLYING THE BIOLOGICAL EFFECTS OF ELECTROMAGNETIC FIELDS; 4. GUIDELINES; 5. CLINICAL USES; CONCLUSION; REFERENCES.

Chapter 4: ELECTROMAGNETIC EFFECTS IN THE NANOSCALE RANGEABSTRACT; 1. CHALLENGES OF LOCAL HYPERTHERMIA; 2. DEEP-HEATING FOR LOCAL-REGIONAL HYPERTHERMIA BY ELECTROMAGNETIC EFFECTS; 3. HOW LOCAL IS THE "LOCAL"?; 4. WHAT DOES ""NON-THERMAL"" MEAN?; 5. SELECTION BY METABOLIC RATE OF CELLS; 6. SELECTION BY DIELECTRIC PROPERTIES OF CELLS AND COMPONENTS; 7. SELECTION BY MORPHOLOGICAL DIFFERENCES OF THE TISSUES; 8. THERMO-ELECTROMAGNETIC EFFECTS AT CELL-MEMBRANES; CONCLUSION; ACKNOWLEDGMENT; REFERENCES; Chapter 5: HSP70 INDUCERS PROTECT AGAINST UVB-INDUCED APOPTOSIS IN KERATINOCYTES; ABSTRACT.

1. INTRODUCTION2. HEAT SHOCK PROTEINS; 3. HSP INDUCERS DERIVED FROM MEDICAL PLANTS PROTECT AGAINST UVB-INDUCED APOPTOSIS; 4. HYPERTHERMIA PROTECTS AGAINST UVB INDUCED APOPTOSIS; CONCLUSION; ACKNOWLEDGMENTS; REFERENCES; Chapter 6: ULTRAVIOLET-INDUCED PHOTOKERATITIS TREATED WITH ASTAXANTHIN; ABSTRACT; 1. INTRODUCTION; 2. MORPHOLOGICAL PROPERTIES -- AST EYEDROPS SHOW PROTECTIVE EFFECTS APPLIED EITHER BEFORE OR AFTER UV EXPOSURE; 3. AST EYEDROPS REDUCED TUNEL-POSITIVE CELLS; 4. AST-TREATED CORNEAS SHOWED LOWER ROS PRODUCTION AND DOWN-REGULATION OF NF-kB EXPRESSION.

5. AST SUPPRESSED PHOTOTOXICITY IN CORNEAL EPITHELIAL CELLS SIMILARLY IN VITROCONCLUSION; ACKNOWLEDGMENTS; REFERENCES; Chapter 7: HEAT-INDUCED GENE EXPRESSION CHANGES IN CANCER CELLS; ABSTRACT; 1. INTRODUCTION; 2. THE EFFECTS OF HEAT STRESS ON CELL DEATH AND HSF1 AND HSPS EXPRESSION; 3. GENES AND BIOLOGICAL FUNCTIONS RESPONSIVE TO HEAT STRESS; 4. GENE NETWORKS AFFECTED BY HEAT STRESS; CONCLUSION; REFERENCES; Chapter 8: HEAT-INDUCED DNA DAMAGE; ABSTRACT; 1. INTRODUCTION; 2. 1980-1999; 3. 2000-2012; 4. RECOGNITION OF HEAT-INDUCED DSBS.

Not all that cannot be seized is an illusion! Many effective powers in nature cannot be seized, including magnetism, electricity and sound. However, they contribute to shaping the quality of human life on a daily basis, transcending health and disease as they interact with the human body. Magnetism is important for stability on earth; however, exposure of the body to magnetic fields is not inert. Such exposure has been reported to alter hormone secretion and stimulate bone formation in vivo. Its coupling with electricity provides light and heat to our planet at certain wavelengths. Yet, the in.

eBooks on EBSCOhost EBSCO eBook Subscription Academic Collection - Worldwide