Early Life Nutrition and Future Health

Connor, Kristin

Early Life Nutrition and Future Health - MDPI - Multidisciplinary Digital Publishing Institute 2020 - 1 electronic resource (182 p.)

Open Access

Inequity starts before birth and is programmed in part by nutritional exposures. If these exposures occur around the time of conception, during pregnancy, and/or in infancy or childhood (all critical periods of development) they may alter a child's health trajectory and impact risk for impaired cognition and learning, and cardiometabolic, immune, and neuropsychiatric diseases and disorders. This Special Issue on "Early Life Nutrition and Future Health" has the following aims: 1) understand the origins of offspring health inequities from an early nutritional perspective; 2) uncover new insights into the environmental, biological, and social mechanisms that underpin these health outcomes in offspring; and 3) present novel targets and approaches to optimise health trajectories and prevent chronic diseases and disorders in later life and across generations. The research projects included herein highlight novel mechanistic, epidemiologic, and intervention studies that target key windows where nutrition has the greatest influence on future health (preconception, prenatal, and postnatal periods) and that explore vulnerable populations and animal models of early life nutritional programming.


Creative Commons


English

books978-3-03928-251-7 9783039282500 9783039282517

10.3390/books978-3-03928-251-7 doi

gut-brain life-course epidemiology milk composition postnatal calcium homeostasis fruit juices phospholipids infant abdominal obesity postpartum L-cell programming gut health development sugars pregnancy gangliosides non-communicable disease prebiotic dietary reference intakes (DRIs) adulthood adult bone health malnutrition supplements nutrition dietary fibre gut barrier child microbiota folic acid supplementation Healthy Eating Index human milk nutrient-sensing signal fetal epidemiology developmental origins of health and disease (DOHaD) dietary intake pH energy intake human milk oligosaccharides undernutrition short-chain fatty acid (SCFA) LC-MS eating behavior gut microbiota reprogramming social inequalities diet quality reduced litter size sphingomyelin oxidative stress

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